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    <title>DSpace コレクション: 1991-06</title>
    <link>http://hdl.handle.net/10564/1975</link>
    <description>1991-06</description>
    <pubDate>Fri, 10 Apr 2026 15:37:12 GMT</pubDate>
    <dc:date>2026-04-10T15:37:12Z</dc:date>
    <item>
      <title>過去8年間における顎骨内囊胞の臨床統計的観察</title>
      <link>http://hdl.handle.net/10564/1986</link>
      <description>タイトル: 過去8年間における顎骨内囊胞の臨床統計的観察
著者: 西岡, 博人; 望月, 光治; 江口, 陽子; 竹内, 尚則; 杉村, 正人; 堀内, 敬介
抄録: Clinico-statistical observation was made in patients with jaw cysts referred to the Department of Oral and Maxillofacial Surgery in the last 8 years. Jaw cysts were classified into odontogenic cysts (475 cases, 81.5%) and non-odontogenic cysts (108 cases, 18.5%). Of odontogenic cysts, radicular cysts were seen in 318 (54.5%), residual cysts in 31 (5.3%), follicular cysts in 90 (15.4%), odontogenic keratocysts in 30 (5.1%), non-keratinized primordial cysts in 5 (0.9%) and calcifying odontogenic cysts in 1 (0.2%), respectively. Of non-odontogenic cysts, incisive canal cysts were seen in 5 (0.9%), globulomaxillary cysts in 4 (0.7%), median palatine cysts in 1 (0.2%), postoperative maxillary cysts in 88 (15.1%), traumatic bone cysts in 7 (1.2%) and static bone cavity in 3 (0.5%), respectively. In follicular cysts, the effect of root canal treatment of deciduous teeth on these cysts&#xD;
was made. Root canal treatment of deciduous teeth was involved in 22 cases (71%) out of&#xD;
31 cases of follicular cysts. In 30 odontogenic keratocysts, dentigerous type was seen in 13 (43%) and primordial type in 17 (57%).</description>
      <pubDate>Sat, 29 Jun 1991 15:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://hdl.handle.net/10564/1986</guid>
      <dc:date>1991-06-29T15:00:00Z</dc:date>
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    <item>
      <title>SLEを伴ったKlinefelter症候群の1例</title>
      <link>http://hdl.handle.net/10564/1985</link>
      <description>タイトル: SLEを伴ったKlinefelter症候群の1例
著者: 泉, 由紀子; 岡本, 新悟; 細川, 彰子; 西山, 利正; 梅本, 典江; 辻井, 正; 村松, 勉; 三馬, 省二; 河田, 陽一; 山田, 一; 岡島, 英五郎
抄録: The case of a 36-year-old male patient with Klinefelter's syndrome associated with systemic lupus erythematosus (SLE) is presented. He was diagnosed as SLE and had been treated since the age of 25 years old. Hypogonadism showing loss of pubic hair and testicular atrophy has appeared since the age of 35. LH-RH test showed a pattern of primary hypogonadism. Karyotype analysis showed that he has 47, XXY chromosomes suggestive of Klinefelter's syndrome. Biopsy of his testis revealed hyaline degeneration of seminiferous tubules and absence of spermatocytes. IgG deposition was found in interstitial cells of testicular tissue by immunohistochemistory. These findings suggest that atrophy of testis is due to immunological changes of SLE as well as hormonal changes of Klinefelter's syndrome.
内容記述: 個人の特定が出来ると思われる写真が掲載されているため本文PDFの登録はなし</description>
      <pubDate>Sat, 29 Jun 1991 15:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://hdl.handle.net/10564/1985</guid>
      <dc:date>1991-06-29T15:00:00Z</dc:date>
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    <item>
      <title>機能性甲状腺癌の一例</title>
      <link>http://hdl.handle.net/10564/1984</link>
      <description>タイトル: 機能性甲状腺癌の一例
著者: 西山, 利正; 岡本, 新悟; 富永, 謙太郎; 細川, 彰子; 梅本, 典江; 泉, 由紀子; 福井, 博; 辻井, 正; 田中, 治; 松永, 喬; 日浅, 義雄
抄録: A 62-year-old woman with large tumor of the thyroid manifesting thyrotoxicosis is presented. Thyroid function tests showed a slight hyperthyroidism. Radiolabelled iodine accumulated in some parts of the tumor and the others were cold. Triiodothyronin (l-T＿3) administration showed non-suppression of the hot pattern. Moreover the blood thyroxin level was not lowered following the suppression of thyrotropin (TSH) induced by&#xD;
l-T＿3 administration. A total tumorectomy was performed and the pathological examination showed various degrees of follicular carcinoma. Histological findings were compared to the radiolabelled iodine uptake by mapping of thyroid scintigram. The most highly accumulated area of radiolabelled iodine consisted of smaller follicles with coloid. However, the unaccumulated areas consisited of cancer cells simulated in fetal thyroid or solid and trabecular patterns. This clinical evidence indicates that thyrotoxicosis in this case is caused by the excessive secretion of thyroid hormones from which were produced the cancer cells of highly accumulated area of radiolabelled iodine.
内容記述: 個人の特定が出来ると思われる写真が掲載されているため本文PDFの登録はなし</description>
      <pubDate>Sat, 29 Jun 1991 15:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://hdl.handle.net/10564/1984</guid>
      <dc:date>1991-06-29T15:00:00Z</dc:date>
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      <title>小児十二指腸Leiomyoblastomaの1例</title>
      <link>http://hdl.handle.net/10564/1983</link>
      <description>タイトル: 小児十二指腸Leiomyoblastomaの1例
著者: 久世, 晋徳; 宮田, 茂樹; 武田, 以知郎; 山下, 隆司; 上辻, 秀和; 山田, 貴; 本郷, 三郎; 富田, 令子; 徳田, 晴厚; 岩垣, 克己
抄録: Leiomyoblastoma, which shows caracteristic pathological features among myogenic tumors, is observed in the uterus as in the whole area of the gastroistestinal tract including the stomach. We recently experienced a case of leiomyoblastoma of the duodenum and here discuss the case with reference to 12 cases previously reported in Japan. A 14-year-old girl with a chief complaint of vomiting was referred to our hospital for further evaluation of an abdominal mass. Upper gastrointestinal X-ray showed a giant tumor on the third portion of the duodenum. A metastatic tumor was recognized in the liver on CT-scan. Duodenectomy was performed. Histology of resected specimen showed Leiomyoblastoma. In the Japanese literature Leiomyoblastoma of the duodemum is rare and has had a greater tendency to be malignant than leiomyoblastoma of the stomach. In our case, the tumor originated in the duodenum and metastatic tumor was observed in the liver. Therefore we considered she should be kept under close medical observation.</description>
      <pubDate>Sat, 29 Jun 1991 15:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://hdl.handle.net/10564/1983</guid>
      <dc:date>1991-06-29T15:00:00Z</dc:date>
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