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  <title>DSpace コレクション: 1997-10</title>
  <link rel="alternate" href="http://hdl.handle.net/10564/1520" />
  <subtitle>1997-10</subtitle>
  <id>http://hdl.handle.net/10564/1520</id>
  <updated>2026-04-09T03:39:27Z</updated>
  <dc:date>2026-04-09T03:39:27Z</dc:date>
  <entry>
    <title>当科における顎骨外骨症の臨床的検討</title>
    <link rel="alternate" href="http://hdl.handle.net/10564/397" />
    <author>
      <name>山本, 一彦</name>
    </author>
    <author>
      <name>馬場, 雅渡</name>
    </author>
    <author>
      <name>北山, 若紫</name>
    </author>
    <author>
      <name>高山, 賢一</name>
    </author>
    <author>
      <name>正田, 晨夫</name>
    </author>
    <author>
      <name>杉村, 正仁</name>
    </author>
    <id>http://hdl.handle.net/10564/397</id>
    <updated>2017-05-29T06:06:57Z</updated>
    <published>1997-10-30T15:00:00Z</published>
    <summary type="text">タイトル: 当科における顎骨外骨症の臨床的検討
著者: 山本, 一彦; 馬場, 雅渡; 北山, 若紫; 高山, 賢一; 正田, 晨夫; 杉村, 正仁
抄録: Two hundred forty cases of exostosis in the oral cavity were clinically &#xD;
investigated. Patients were 57 males and 183 females and mostly in their 4 th to 6 th decades &#xD;
of age. Torus palatinus was observed in 143 patients and torus mandibularis in 126 patients. &#xD;
Symptoms such as pain and abnormal sensation were found in 38% of patients. Torus &#xD;
palatinus were mostly 15-25 mm in size and morphologically flat and spindle types. Three &#xD;
fourths of torus mandibularis were bilaterally observed and multiple type was found more &#xD;
frequently than single type. Twenty-five percents of torus palatinus were accompanied by &#xD;
torus mandibularis. Surgical excision was performed in 25 cases, mostly in cases with &#xD;
symptoms.</summary>
    <dc:date>1997-10-30T15:00:00Z</dc:date>
  </entry>
  <entry>
    <title>先天性絞扼輪症候群の2例</title>
    <link rel="alternate" href="http://hdl.handle.net/10564/396" />
    <author>
      <name>内田, 優美子</name>
    </author>
    <author>
      <name>西久保, 敏也</name>
    </author>
    <author>
      <name>箕輪, 秀樹</name>
    </author>
    <author>
      <name>野上, 恵嗣</name>
    </author>
    <author>
      <name>上辻, 秀和</name>
    </author>
    <author>
      <name>下山, 弘展</name>
    </author>
    <author>
      <name>吉田, 裕慈</name>
    </author>
    <author>
      <name>川口, 千晴</name>
    </author>
    <author>
      <name>中山, 雅博</name>
    </author>
    <author>
      <name>高橋, 幸博</name>
    </author>
    <author>
      <name>吉岡, 章</name>
    </author>
    <id>http://hdl.handle.net/10564/396</id>
    <updated>2017-05-29T06:06:49Z</updated>
    <published>1997-10-30T15:00:00Z</published>
    <summary type="text">タイトル: 先天性絞扼輪症候群の2例
著者: 内田, 優美子; 西久保, 敏也; 箕輪, 秀樹; 野上, 恵嗣; 上辻, 秀和; 下山, 弘展; 吉田, 裕慈; 川口, 千晴; 中山, 雅博; 高橋, 幸博; 吉岡, 章
抄録: We encountered two cases of congenital constriction band syndrome &#xD;
(CCBS). The first case was 11-day-old boy weighing 3,150g delivered at 38 weeks of &#xD;
gestational age. He revealed constriction band on the proximal part of the right index, &#xD;
digitus medius and digitus anularis. These fingers were shortened and syndactyly between &#xD;
digitus medius and digitus anularis was present with fenestration. The second case was a &#xD;
0-day-old girl weighing 1,590g delivered at 36 weeks of gestational age. Fetal echography &#xD;
revealed decreased amniotic fluid and intrauterine growth retardation at 36 gastational &#xD;
weeks. She had a constriction band of the left wrist joint with deformity of the left hand. &#xD;
Both infants were diagnosed with congenital constriction band syndrome based on the &#xD;
presence of a typical constriction band, without any other anomaly. Therefore, disruption &#xD;
of the amniotic membrane was speculated to have occurred at 5 to 7 weeks of gestational &#xD;
age.</summary>
    <dc:date>1997-10-30T15:00:00Z</dc:date>
  </entry>
  <entry>
    <title>不明熱と高IgM血症を呈した悪性リンパ腫の1剖検例</title>
    <link rel="alternate" href="http://hdl.handle.net/10564/395" />
    <author>
      <name>高井, 英子</name>
    </author>
    <author>
      <name>山野, 繁</name>
    </author>
    <author>
      <name>勝山, 慶之</name>
    </author>
    <author>
      <name>松田, 尚史</name>
    </author>
    <author>
      <name>椎木, 英夫</name>
    </author>
    <author>
      <name>藤井, 謙裕</name>
    </author>
    <author>
      <name>土肥, 和紘</name>
    </author>
    <author>
      <name>遠藤, 武弘</name>
    </author>
    <author>
      <name>小西, 陽一</name>
    </author>
    <author>
      <name>中村, 忍</name>
    </author>
    <id>http://hdl.handle.net/10564/395</id>
    <updated>2017-05-29T06:06:53Z</updated>
    <published>1997-10-30T15:00:00Z</published>
    <summary type="text">タイトル: 不明熱と高IgM血症を呈した悪性リンパ腫の1剖検例
著者: 高井, 英子; 山野, 繁; 勝山, 慶之; 松田, 尚史; 椎木, 英夫; 藤井, 謙裕; 土肥, 和紘; 遠藤, 武弘; 小西, 陽一; 中村, 忍
抄録: We report a case of an 80-year-old man with non-Hodgkin's lymphoma and &#xD;
high serum level of IgM. He was admitted to our hospital because of fever of unknown &#xD;
etiology and hoarseness. Seven weeks before the current admission, the patient was &#xD;
admitted to another hospital because of high fever, cough, and anasarca on January 26, 1994. &#xD;
High serum level of IgM was detected by immunoelectrophoresis in February 1994. &#xD;
Laboratory tests on admission revealed leucopenia (2,200/μl), high serum level of LDH &#xD;
(1,012IU/l) and IgM (1,509mg/dl). An abdominal computed tomographic (CT) scan &#xD;
revealed hepatosplenomegaly without lymphadenopathy.&#xD;
The patient was diagnosed as having non-Hodgkin's lymphoma (small lymphocytic + &#xD;
plasmacytoid) by autopsy. Lymphadenopathy was not detected, but tumor cells were &#xD;
microscopically detected in jugular, axillary, mediastinal, and adbominal lymphnodes. Also &#xD;
tumor cells had infiltrated to the liver, kidneys, spleen, and pancreas. Immunohistochemical &#xD;
staining for anti-IgM, IgG, κ-light chain, and λ-light chain showed positive in lymphoid &#xD;
cells. We experienced a difficult case to diagnose as malignant lymphoma for lack of &#xD;
lymphadenopathy.</summary>
    <dc:date>1997-10-30T15:00:00Z</dc:date>
  </entry>
  <entry>
    <title>RESPONSE TO THE FUROSEMIDE TEST MAY PREDICT THE EFFECTS OF DELAPRIL ON RENAL FUNCTION IN PATIENTS WITH CHRONIC RENAL INSUFFICIENCY</title>
    <link rel="alternate" href="http://hdl.handle.net/10564/394" />
    <author>
      <name>Fujii, Yoshihiro</name>
    </author>
    <author>
      <name>Kishimoto, Tadashi</name>
    </author>
    <author>
      <name>Dohi, Kazuhiro</name>
    </author>
    <id>http://hdl.handle.net/10564/394</id>
    <updated>2017-06-11T23:20:26Z</updated>
    <published>1997-10-30T15:00:00Z</published>
    <summary type="text">タイトル: RESPONSE TO THE FUROSEMIDE TEST MAY PREDICT THE EFFECTS OF DELAPRIL ON RENAL FUNCTION IN PATIENTS WITH CHRONIC RENAL INSUFFICIENCY
著者: Fujii, Yoshihiro; Kishimoto, Tadashi; Dohi, Kazuhiro
抄録: Background : It is not clear whether the administration of an angiotensin- &#xD;
converting enzyme (ACE) inhibitor consisitently slows the progression of disease in &#xD;
patients with renal disease. We investigated the usefulness of the furosemide test in &#xD;
predicting the efficacy of delapril, an ACE inhibitor, in patients with chronic renal insuffi- &#xD;
clency.&#xD;
Methods : Delapril (7.5 mg to 15 mg) was administered daily to 24 patients with chronic &#xD;
renal insufficiency as indicated by a serum concentration of creatinine (Scr) between 1.2 &#xD;
mg/dl and 3.0 mg/dl. Patients were classified into improved, unchanged, and worsened &#xD;
groups based on the effect of delapril on the Scr. We measured the following parameters &#xD;
before the administration of delapril for a mean of 9 months blood pressure, Scr, urinary &#xD;
protein excretion, urinary sodium excretion, and changes in the renin and aldosterone &#xD;
response (the PRA ratio and PAC ratio) to a furosemide test.&#xD;
Results : Renal function was improved in 6 patients, unchanged in 8 patients, and worsend &#xD;
in 10 patients after delapril treatment. Urinary protein excretion was decreased, but not &#xD;
significantly, in all three groups. No significant differences in baseline parameters were &#xD;
observed among the groups before treatment. The PRA ratio was significantly higher in the &#xD;
group showing improvement vs. the groups showing either no improvement or a worsening &#xD;
of renal function.&#xD;
Conclusions : Delapril tended to reduce the urinary excretion of protein in patients with &#xD;
chronic renal insufficienty. However, delapril did not consistently reduce the rate of decline &#xD;
in glomerular filtration rate. The protective effect of this drug on renal function appeared &#xD;
to be related to the renin response to furosemide test.</summary>
    <dc:date>1997-10-30T15:00:00Z</dc:date>
  </entry>
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